{"id":838,"date":"2023-08-10T15:23:11","date_gmt":"2023-08-10T15:23:11","guid":{"rendered":"https:\/\/pediatricbrainfoundation.org\/archive\/autism-spectrum-disorders-page-5\/"},"modified":"2023-08-10T15:23:11","modified_gmt":"2023-08-10T15:23:11","slug":"autism-spectrum-disorders-page-5","status":"publish","type":"page","link":"https:\/\/pediatricbrainfoundation.org\/archive\/autism-spectrum-disorders-page-5\/","title":{"rendered":"Autism Spectrum Disorders"},"content":{"rendered":"
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Autism Spectrum Disorders<\/h1>\n<\/section><\/div>\n
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Social symptoms in autistic children may be caused by hyper-connected neurons<\/a><\/h2>\n
\n Thursday, November 7, 2013<\/span>  \u00b7  Posted by Cell Press<\/a> <\/div>\n

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The findings could lead to new treatment strategies and new ways to detect autism early, the researchers say. Autism spectrum disorder is a neurodevelopmental condition affecting nearly 1 in 88 children.<\/div>\n<\/p><\/div>\n<\/div>\n<\/article>\n

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Earliest marker for autism found in young infants<\/a><\/h2>\n
\n Wednesday, November 6, 2013<\/span>  \u00b7  Posted by NIH\/National Institutes of Health<\/a> <\/div>\n

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Eye contact during early infancy may be a key to early identification of autism, according to a study funded by the National Institute of Mental Health (NIMH), part of the National Institutes of Health. Published this week in the journal Nature, the study reveals the earliest sign of developing autism ever observed \u2014 a steady decline in attention to others\u2019 eyes within the first two to six months of life.<\/div>\n<\/p><\/div>\n<\/div>\n<\/article>\n

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Gene found to foster synapse formation in the brain<\/a><\/h2>\n
\n Thursday, October 31, 2013<\/span>  \u00b7  Posted by Johns Hopkins Medicine<\/a> <\/div>\n

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Researchers at Johns Hopkins say they have found that a gene already implicated in human speech disorders and epilepsy is also needed for vocalizations and synapse formation in mice. The finding, they say, adds to scientific understanding of how language develops, as well as the way synapses \u2014 the connections among brain cells that enable us to think \u2014 are formed.<\/div>\n<\/p><\/div>\n<\/div>\n<\/article>\n

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Autism and Language Impairment Genetically Linked<\/a><\/h2>\n
\n Wednesday, October 30, 2013<\/span>  \u00b7  Posted by Rutgers University<\/a> <\/div>\n

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New research by scientists at Rutgers University and The Research Institute at Nationwide Children\u2019s Hospital in Ohio, reveals that there is a genetic link connecting family members with autism like Lorenzo Miodus-Santini to those like his brother, Christian, who have specific language impairment characterized by speech and language difficulties that can\u2019t be explained by cognitive or physical problems.<\/div>\n<\/p><\/div>\n<\/div>\n<\/article>\n

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New study examines link between pregnancy weight gain, autism spectrum disorders<\/a><\/h2>\n
\n Monday, October 28, 2013<\/span>  \u00b7  Posted by University of Utah Health Sciences<\/a> <\/div>\n

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Researchers have uncovered an association between autism spectrum disorders and a small increase in the amount of weight a mother gains during pregnancy. <\/div>\n<\/p><\/div>\n<\/div>\n<\/article>\n

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UNC child neurologist finds potential route to better treatments for Fragile X, autism<\/a><\/h2>\n
\n Wednesday, October 23, 2013<\/span>  \u00b7  Posted by University of North Carolina School of Medicine <\/a> <\/div>\n

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Malanga\u2019s finding suggests that not all people with Fragile X share the same biological hurdles. The same is likely true, he said, for people with other autism-related disorders, such as Rett syndrome and Angelman syndrome.<\/div>\n<\/p><\/div>\n<\/div>\n<\/article>\n

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Two genetic wrongs make a biochemical right<\/a><\/h2>\n
\n Sunday, October 20, 2013<\/span>  \u00b7  Posted by University of Massachusetts Medical School<\/a> <\/div>\n

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The prime cause of the Fragile X syndrome may be a translational imbalance that results in elevated protein production in the brain. Restoration of this balance may be necessary for normal neurological function. <\/div>\n<\/p><\/div>\n<\/div>\n<\/article>\n

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LSUHSC bird study finds key info about human speech-language development<\/a><\/h2>\n
\n Thursday, October 17, 2013<\/span>  \u00b7  Posted by Louisiana State University Health Sciences Center<\/a> <\/div>\n

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Understanding how miRNAs regulate FOXP2 may open many possibilities to influence speech and language development through genetic variations in miRNA genes, as well as behavioral and environmental factors.<\/div>\n<\/p><\/div>\n<\/div>\n<\/article>\n

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The pig, the fish, and the jellyfish: Tracing nervous disorders in humans<\/a><\/h2>\n
\n Wednesday, October 16, 2013<\/span>  \u00b7  Posted by Aarhus University<\/a> <\/div>\n

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The pig, the jellyfish and the zebrafish are being used by scientists at Aarhus University to, among other things, gain a greater understanding of hereditary forms of diseases affecting the nervous system. This can be disorders like Parkinson\u2019s disease, Alzheimer\u2019s disease, autism, epilepsy and the motor neurone disease ALS.<\/div>\n<\/p><\/div>\n<\/div>\n<\/article>\n

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Rett Syndrome Gene Dysfunction Redefined<\/a><\/h2>\n
\n Thursday, October 3, 2013<\/span>  \u00b7  Posted by Whitehead Institute for Biomedical Research <\/a> <\/div>\n

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“Now we have a much better understanding of the function of MECP2, and the severity of the disease on a cellular level. Knowing that human Rett neurons are impaired in both global transcription and translation is important for us to design therapeutic strategies for Rett. Growth factors such as BDNF and IGF-1 are known to activate the AKT\/mTOR pathway and increase protein synthesis. Down the road, we are interested in further exploring the Akt\/mTOR pathway, and investigate how activation of this pathway could reverse the disease.\u201d<\/div>\n<\/p><\/div>\n<\/div>\n<\/article>\n

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