{"id":1083,"date":"2023-08-10T15:23:31","date_gmt":"2023-08-10T15:23:31","guid":{"rendered":"https:\/\/pediatricbrainfoundation.org\/archive\/memory-loss\/"},"modified":"2023-08-10T15:23:31","modified_gmt":"2023-08-10T15:23:31","slug":"memory-loss","status":"publish","type":"page","link":"https:\/\/pediatricbrainfoundation.org\/archive\/memory-loss\/","title":{"rendered":"Memory loss"},"content":{"rendered":"
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International Research Team Discovers Genetic Dysfunction Connected to Hydrocephalus<\/a><\/h2>\n
\n Thursday, July 17, 2014<\/span>  \u00b7  Posted by Case Western Reserve University <\/a> <\/div>\n

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The mysterious condition once known as \u201cwater on the brain\u201d became just a bit less murky this week thanks to a global research group led in part by a Case Western Reserve researcher. Professor Anthony Wynshaw-Boris, MD, PhD, is the co-principal investigator on a study that illustrates how the domino effect of one genetic error can contribute to excessive cerebrospinal fluid surrounding the brains of mice \u2014 a disorder known as hydrocephalus.
\nScientists are still at the most nascent stages of understanding different causes and kinds of hydrocephalus. In some instances, the root sources are genetic; in others, the fluid accumulation is attributed to complications of premature birth. This project illuminates one way in which genetic influences contribute to the condition.<\/div>\n<\/p><\/div>\n<\/div>\n<\/article>\n

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New Hope for Victims of Traumatic Brain Injury<\/a><\/h2>\n
\n Monday, November 18, 2013<\/span>  \u00b7  Posted by American Friends of Tel Aviv University <\/a> <\/div>\n

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Evidence that hyperbaric oxygen therapy (HBOT) should repair chronically impaired brain functions and significantly improve the quality of life of mild TBI patients. The new findings challenge the often-dismissive stand of the US Food and Drug Administration, Centers for Disease Control and Prevention, and the medical community at large, and offer new hope where there was none.<\/div>\n<\/p><\/div>\n<\/div>\n<\/article>\n

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Stanford senior a pioneer in traumatic brain injury research<\/a><\/h2>\n
\n Tuesday, November 12, 2013<\/span>  \u00b7  Posted by Stanford University<\/a> <\/div>\n

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A biology student at Stanford and researchers at the National Institutes of Health have devised a method for observing the immediate effects of a mild traumatic brain injury (TBI) in real time in mice. The work has revealed how individual cells respond to the injury and has helped the researchers suggest a possible therapeutic approach for limiting brain damage in humans.<\/div>\n<\/p><\/div>\n<\/div>\n<\/article>\n

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Football’s Concussion Problem, in 3 Terrifying Pictures<\/a><\/h2>\n
\n Tuesday, October 8, 2013<\/span>  \u00b7  Posted by Mother Jones<\/a> <\/div>\n

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While the symptoms of a concussion\u2014dizziness, vomiting, memory loss\u2014can be felt immediately, the long-term impacts of repeated brain trauma have been harder to study. Research points to chronic traumatic encephalopathy, or CTE, as one of the major outcomes. CTE is caused by a buildup of tau, a protein that strangles brain cells and degenerates brain tissue, which is caused by repetitive brain trauma like the hits football players endure.<\/div>\n<\/p><\/div>\n<\/div>\n<\/article>\n

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Scientists reverse memory loss in animal brain cells<\/a><\/h2>\n
\n Tuesday, April 16, 2013<\/span>  \u00b7  Posted by University of Texas Health Science Center at Houston<\/a> <\/div>\n

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This methodology may apply to humans if we can identify the same biochemical processes in humans. Our results suggest a new strategy for treatments of cognitive impairment. Mathematical models might help design therapies that optimize the combination of training protocols with traditional drug treatments, Byrne said.<\/div>\n<\/p><\/div>\n<\/div>\n<\/article>\n

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Protein regulation linked to intellectual disability<\/a><\/h2>\n
\n Thursday, October 25, 2012<\/span>  \u00b7  Posted by University of Adelaide <\/a> <\/div>\n

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While many intellectual disabilities are caused directly by a genetic mutation in the so-called “protein coding” part of our genes, the researchers found that in their case the answer laid outside the gene and in the regulation of proteins.<\/div>\n<\/p><\/div>\n<\/div>\n<\/article>\n

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